Víctor Cilleros Mañé

Work

Institute for Bioengineering of Catalonia
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PostDoc position

Spain

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Universitat Rovira i Virgili
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Postdoctoral researcher

Spain

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Publications

Protective effects of grape seed procyanidin extract on neurotrophic and muscarinic signaling pathways in the aging neuromuscular junction

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Food & Function

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Molecular Adaptations of BDNF/NT-4 Neurotrophic and Muscarinic Pathways in Ageing Neuromuscular Synapses

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International Journal of Molecular Sciences

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Molecular Adaptations of BDNF/NT-4 Neurotrophic and Muscarinic Pathways in Ageing Neuromuscular Synapses

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International Journal of Molecular Sciences

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BDNF/TrkB signalling, in cooperation with muscarinic signalling, retrogradely regulates PKA pathway to phosphorylate SNAP-25 and Synapsin-1 at the neuromuscular junction

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Cell Communication and Signaling

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Synaptic retrograde regulation of the PKA-induced SNAP-25 and Synapsin-1 phosphorylation

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Cellular & Molecular Biology Letters

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Muscarinic Receptors in Developmental Axonal Competition at the Neuromuscular Junction

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Molecular Neurobiology

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Synaptic retrograde regulation of the PKA-induced SNAP-25 and Synapsin-1 phosphorylation

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Cellular and Molecular Biology Letters

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Brain-derived neurotrophic factor signaling in the neuromuscular junction during developmental axonal competition and synapse elimination

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Neural Regeneration Research

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Synaptic retrograde regulation of the PKA-induced SNAP-25 and Synapsin-1 phosphorylation

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Cellular & Molecular Biology Letters

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TrkB signaling is correlated with muscular fatigue resistance and less vulnerability to neurodegeneration

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Frontiers in Molecular Neuroscience

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Muscarinic Receptors in Developmental Axonal Competition at the Neuromuscular Junction

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Molecular Neurobiology

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Involvement of the Voltage-Gated Calcium Channels L- P/Q- and N-Types in Synapse Elimination During Neuromuscular Junction Development

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Molecular Neurobiology

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Involvement of the Voltage-Gated Calcium Channels L- P/Q- and N-Types in Synapse Elimination During Neuromuscular Junction Development

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Molecular Neurobiology

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TrkB signaling is correlated with muscular fatigue resistance and less vulnerability to neurodegeneration

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Frontiers in Molecular Neuroscience

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TrkB signaling is correlated with muscular fatigue resistance and less vulnerability to neurodegeneration

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Frontiers in Molecular Neuroscience

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PKA and PKC Balance in Synapse Elimination during Neuromuscular Junction Development

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Cells

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M1 and M2 mAChRs activate PDK1 and regulate PKC βI and ε and the exocytotic apparatus at the NMJ

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The FASEB Journal

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M<sub>1</sub> and M<sub>2</sub> mAChRs activate PDK1 and regulate PKC βI and ε and the exocytotic apparatus at the NMJ

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FASEB Journal

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M 1 and M 2 mAChRs activate PDK1 and regulate PKC βI and ε and the exocytotic apparatus at the NMJ

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The FASEB Journal

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Running and Swimming Differently Adapt the BDNF/TrkB Pathway to a Slow Molecular Pattern at the NMJ

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International Journal of Molecular Sciences

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Running and swimming differently adapt the bdnf/trkb pathway to a slow molecular pattern at the nmj

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International Journal of Molecular Sciences

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PKA and PKC balance in synapse elimination during neuromuscular junction development

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Cells

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PKA and PKC Balance in Synapse Elimination during Neuromuscular Junction Development

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Cells

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The M2 muscarinic receptor, in association to M1, regulates the neuromuscular PKA molecular dynamics

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The FASEB Journal

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The M 2 muscarinic receptor, in association to M 1 , regulates the neuromuscular PKA molecular dynamics

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The FASEB Journal

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The M<inf>2</inf> muscarinic receptor, in association to M<inf>1</inf>, regulates the neuromuscular PKA molecular dynamics

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FASEB Journal

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Running and swimming prevent the deregulation of the BDNF/TrkB neurotrophic signalling at the neuromuscular junction in mice with amyotrophic lateral sclerosis

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Cellular and Molecular Life Sciences

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Running and swimming prevent the deregulation of the BDNF/TrkB neurotrophic signalling at the neuromuscular junction in mice with amyotrophic lateral sclerosis

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Cellular and Molecular Life Sciences

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Overview of Impaired BDNF Signaling, Their Coupled Downstream Serine-Threonine Kinases and SNARE/SM Complex in the Neuromuscular Junction of the Amyotrophic Lateral Sclerosis Model SOD1-G93A Mice

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Molecular Neurobiology

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The Impact of Kinases in Amyotrophic Lateral Sclerosis at the Neuromuscular Synapse: Insights into BDNF/TrkB and PKC Signaling

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Cells

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The impact of kinases in amyotrophic lateral sclerosis at the neuromuscular synapse: Insights into BDNF/TRKB and PKC signaling

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Cells

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Opposed Actions of PKA Isozymes (RI and RII) and PKC Isoforms (cPKCβI and nPKCε) in Neuromuscular Developmental Synapse Elimination

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Cells

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Opposed actions of PKA isozymes (RI and RII) and PKC isoforms (cPKCβI and nPKCε) in neuromuscular developmental synapse elimination

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Cells

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Running and swimming prevent the deregulation of the BDNF/TrkB neurotrophic signalling at the neuromuscular junction in mice with amyotrophic lateral sclerosis

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Cellular and Molecular Life Sciences

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Overview of Impaired BDNF Signaling, Their Coupled Downstream Serine-Threonine Kinases and SNARE/SM Complex in the Neuromuscular Junction of the Amyotrophic Lateral Sclerosis Model SOD1-G93A Mice

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Molecular Neurobiology

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nPKCε Mediates SNAP-25 Phosphorylation of Ser-187 in Basal Conditions and After Synaptic Activity at the Neuromuscular Junction

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Molecular Neurobiology

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nPKCε Mediates SNAP-25 Phosphorylation of Ser-187 in Basal Conditions and After Synaptic Activity at the Neuromuscular Junction

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Molecular Neurobiology

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Axonal competition and synapse elimination during neuromuscular junction development

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Current Opinion in Physiology

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BDNF-TrkB Signaling Coupled to nPKCε and cPKCβI Modulate the Phosphorylation of the Exocytotic Protein Munc18-1 During Synaptic Activity at the Neuromuscular Junction

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Frontiers in Molecular Neuroscience

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BDNF-TrKB signaling coupled to nPKCε and cPKCβI modulate the phosphorylation of the exocytotic protein MUNC18-1 during synaptic activity at the neuromuscular junction

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Frontiers in Molecular Neuroscience

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Adenosine receptors in developing and adult mouse neuromuscular junctions and functional links with other metabotropic receptor pathways

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Frontiers in Pharmacology

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Adenosine Receptors in Developing and Adult Mouse Neuromuscular Junctions and Functional Links With Other Metabotropic Receptor Pathways

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Frontiers in Pharmacology

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Axonal competition and synapse elimination during neuromuscular junction development

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Current Opinion in Physiology

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Muscle contraction regulates bdnf/trkb signaling to modulate synaptic function through presynaptic cPKCα and cPKCβi

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Frontiers in Molecular Neuroscience

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Presynaptic Membrane Receptors Modulate ACh Release, Axonal Competition and Synapse Elimination during Neuromuscular Junction Development

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Frontiers in Molecular Neuroscience

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Presynaptic membrane receptors modulate ACh release, axonal competition and synapse elimination during neuromuscular junction development

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Frontiers in Molecular Neuroscience

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Presynaptic Muscarinic Acetylcholine Receptors and TrkB Receptor Cooperate in the Elimination of Redundant Motor Nerve Terminals during Development

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Frontiers in Aging Neuroscience

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Presynaptic muscarinic acetylcholine receptors and TrkB receptor cooperate in the elimination of redundant motor nerve terminals during development

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Frontiers in Aging Neuroscience

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Synergistic Action of Presynaptic Muscarinic Acetylcholine Receptors and Adenosine Receptors in Developmental Axonal Competition at the Neuromuscular Junction

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Developmental Neuroscience

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Synaptic Activity and Muscle Contraction Increases PDK1 and PKCβI Phosphorylation in the Presynaptic Membrane of the Neuromuscular Junction

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Frontiers in Molecular Neuroscience

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Synaptic activity and muscle contraction increases PDK1 and PKCβi phosphorylation in the presynaptic membrane of the neuromuscular junction

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Frontiers in Molecular Neuroscience

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Membrane Receptor-Induced Changes of the Protein Kinases A and C Activity May Play a Leading Role in Promoting Developmental Synapse Elimination at the Neuromuscular Junction

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Frontiers in Molecular Neuroscience

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Membrane receptor-induced changes of the protein kinases A and C activity may play a leading role in promoting developmental synapse elimination at the neuromuscular junction

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Frontiers in Molecular Neuroscience

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Muscle Contraction Regulates BDNF/TrkB Signaling to Modulate Synaptic Function through Presynaptic cPKCα and cPKCβI

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Frontiers in Molecular Neuroscience

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Synergistic Action of Presynaptic Muscarinic Acetylcholine Receptors and Adenosine Receptors in Developmental Axonal Competition at the Neuromuscular Junction

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Developmental Neuroscience

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