Tomoki Inagaki

Work

Fujita Health University
|

Assistant professor

Japan

University of California, Davis
|

Postdoctoral fellow

US

Chubu Rosai Hospital
|

Resident

Japan

Education

Nagoya University
Japan

Ph.D.

Nagoya University
Japan

M.D.

Publications

Studies on Gene Enhancer with KSHV mini-chromatin

Summary

preprint

Design, development, and evaluation of gene therapeutics specific to KSHV-associated diseases

Summary

preprint

The Role of vIL-6 in KSHV-Mediated Immune Evasion and Tumorigenesis

Published by

Viruses

Summary

journal-article

The Role of vIL-6 in KSHV-Mediated Immune Evasion and Tumorigenesis

Published by

Viruses

Summary

journal-article

A LANA peptide inhibits tumor growth by inducing CHD4 protein cleavage and triggers cell death

Published by

Cell Chemical Biology

Summary

journal-article

An atlas of chromatin landscape in KSHV-infected cells during de novo infection and reactivation

Published by

Virology

Summary

journal-article

Kaposi’s sarcoma-associated herpesvirus terminal repeat regulates inducible lytic gene promoters

Published by

Journal of Virology

Summary

journal-article

Epstein-Barr virus lytic gene BNRF1 promotes B-cell lymphomagenesis via IFI27 upregulation

Published by

PLOS Pathogens

Summary

journal-article

KSHV vIL-6 enhances inflammatory responses by epigenetic reprogramming

Published by

PLOS Pathogens

Summary

journal-article

Virally encoded interleukin-6 facilitates KSHV replication in monocytes and induction of dysfunctional macrophages

Published by

PLOS Pathogens

Summary

journal-article

Kaposi’s Sarcoma-Associated Herpesvirus (KSHV) LANA Prevents KSHV Episomes from Degradation

Summary

preprint

KSHV Topologically Associating Domains in Latent and Reactivated Viral Chromatin

Published by

Journal of Virology

Summary

journal-article

KSHV episome tethering sites on host chromosomes and regulation of latency-lytic switch by CHD4

Published by

Cell Reports

Summary

journal-article

Molecular Basis of Epstein–Barr Virus Latency Establishment and Lytic Reactivation

Published by

Viruses

Summary

journal-article

Direct Evidence of Abortive Lytic Infection-Mediated Establishment of Epstein-Barr Virus Latency During B-Cell Infection

Published by

Frontiers in Microbiology

Summary

journal-article

S-Like-Phase Cyclin-Dependent Kinases Stabilize the Epstein-Barr Virus BDLF4 Protein To Temporally Control Late Gene Transcription

Published by

Journal of Virology

Summary

journal-article